Investigative Ophthalmology & Visual Science Cover Image for Volume 65, Issue 7
June 2024
Volume 65, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2024
Increased susceptibility to myopia in Prkca-/- mice, which lack PKCα, a marker of rod bipolar cells
Author Affiliations & Notes
  • Robert Duvoisin
    Chemical Physiology & Biochemistry, Oregon Health & Science University, Portland, Oregon, United States
  • Wilmet Baptiste
    Institut de la vision, Sorbonne Université, INSERM, CNRS, Paris, France
  • Tammie Haley
    Chemical Physiology & Biochemistry, Oregon Health & Science University, Portland, Oregon, United States
  • Jacques Callebert
    Biochemistry and Molecular Biology, INSERM U942, Hopital Lariboisière, Paris, France
  • Christina Zeitz
    Institut de la vision, Sorbonne Université, INSERM, CNRS, Paris, France
  • Catherine W Morgans
    Chemical Physiology & Biochemistry, Oregon Health & Science University, Portland, Oregon, United States
  • Footnotes
    Commercial Relationships   Robert Duvoisin None; Wilmet Baptiste None; Tammie Haley None; Jacques Callebert None; Christina Zeitz None; Catherine Morgans None
  • Footnotes
    Support  NIH grant EY029985 (RD and CM); IRP-INSERM (CZ and RD); ANR-chair with Essilor: MYOPIAMASTER (CZ); Retina France, Association Valentin Haüy (CZ).
Investigative Ophthalmology & Visual Science June 2024, Vol.65, 3927. doi:
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    • Get Citation

      Robert Duvoisin, Wilmet Baptiste, Tammie Haley, Jacques Callebert, Christina Zeitz, Catherine W Morgans; Increased susceptibility to myopia in Prkca-/- mice, which lack PKCα, a marker of rod bipolar cells. Invest. Ophthalmol. Vis. Sci. 2024;65(7):3927.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : High myopia is a major risk factor for blinding ophthalmic diseases, yet the genes and mechanisms that give rise to it are poorly understood. Complete congenital stationary night blindness (cCSNB) is caused by mutations in genes expressed in ON-bipolar cells and is strongly correlated with high myopia. To gain insight into molecular mechanisms of myopia, whole transcriptome sequencing (RNA-seq) was performed on three mouse models of cCNSB (Zeitz et al. Prog Ret Eye Res. 2023). Prkca, which encodes PKCα, a marker of rod bipolar cells, was found to be differentially expressed in all three cCSNB models. While Prkca has not been previously shown to be associated with high myopia, because of its differential upregulation, we hypothesized that Prkca knockout mice would have reduced levels of retinal dopamine and an increased susceptibility to lens-induced myopia (LIM).

Methods : Prkca +/+ and Prkca-/- mice were used in this study. Retinal levels of dopamine and its metabolite 3,4 dihydroxyphenylacetic acid (DOPAC) were measured using ultra performance liquid chromatography. Myopia was induced by placing a -25 D lens in front of the right eye of 3-week-old mice for 3 weeks (lens-induced myopia, LIM). Refractive development and LIM optic shift were measured using an infrared photorefractor, as developed by Frank Schaeffel (Optom Vis Sci. 2004). Full field electroretinograms (ERGs) were performed on both LIM and control eyes in wild type and Prkca-/- mice. Oscillatory potentials (OPs) were obtained by 30-150 Hz bandpass filtering.

Results : Dopamine and DOPAC levels were reduced in Prkca-/- mice compared to wild type. Refraction measurements in 3- and 4-week-old untreated mice revealed that Prkca-/- eyes were myopic compared to wild type eyes, but recovered and were similar and slightly hyperopic at 6 weeks of age and older. After 3 weeks of LIM, preliminary data showed a larger myopic shift in Prkca-/- eyes compared to wild type. ERG a- and b-waves were not significantly different between LIM and control eyes. Normalized to the b-wave, OPs appear to be larger in recordings from LIM eyes, compared to control.

Conclusions : Our results indicate that PKCα, which is abundantly expressed in rod bipolar cells, may be important for emmetropization. This further reinforces the contribution of the retinal ON pathway in regulating axial growth of the eye.

This abstract was presented at the 2024 ARVO Annual Meeting, held in Seattle, WA, May 5-9, 2024.

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