Investigative Ophthalmology & Visual Science Cover Image for Volume 65, Issue 7
June 2024
Volume 65, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2024
Integration of Hypoimmunogenic Human Retinal Ganglion Cells in Rat Retina: Role of Chemotropic Molecules
Author Affiliations & Notes
  • Murali Subramani
    Ophthalmology and Visual Science, University of Nebraska Medical Center College of Medicine, Omaha, Nebraska, United States
  • Brandon Lambrecht
    Ophthalmology and Visual Science, University of Nebraska Medical Center College of Medicine, Omaha, Nebraska, United States
  • Mahendra Rao
    CDO, Vita Therapeutics, Baltimore, Maryland, United States
  • Iqbal Ahmad
    Ophthalmology and Visual Science, University of Nebraska Medical Center College of Medicine, Omaha, Nebraska, United States
  • Footnotes
    Commercial Relationships   Murali Subramani None; Brandon Lambrecht None; Mahendra Rao None; Iqbal Ahmad None
  • Footnotes
    Support  NIH Grant EY022051, EY029778
Investigative Ophthalmology & Visual Science June 2024, Vol.65, 3328. doi:
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      Murali Subramani, Brandon Lambrecht, Mahendra Rao, Iqbal Ahmad; Integration of Hypoimmunogenic Human Retinal Ganglion Cells in Rat Retina: Role of Chemotropic Molecules. Invest. Ophthalmol. Vis. Sci. 2024;65(7):3328.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : The success of ex-vivo stem cell approach to glaucomatous degeneration requires optimal integration of transplanted retinal ganglion cells (RGCs) without immune rejection in the host retina. There is a knowledge gap about the influence of chemotropic environment on the integration of RGCs and to address the immune rejection. Here, we have generated human RGCs from hypoimmunogenic human induced pluripotent stem cells (hiPSCs) and examined if they could survive without immune suppression following transplantation and incorporate better upon targeted manipulation of chemotropic molecules in the host retina.

Methods : RGCs were directly differentiated from engineered human hypoimmunogenic hiPSCs and hESCs by recapitulating developmental mechanism. hiPSC/hES-RGCs were transplanted in PN10 rat retinal explants, pretreated with chondroitinase to reduce chondroitin sulfate proteoglycans (CSPGs)-mediated chemo-repulsion. hiPSC/hES-RGCs were preincubated with anti-ROBO2 antibody to prevent chemo-repulsive response to Slits. Media included Netrin-1 and SDF-1 as chemo-attractants. Controls included untreated hRGCs and explants. ROBO2 neutralized hiPSC-RGCs were transplanted intravitreally in PN10 rats without an immune suppressant.

Results : We observed that hRGCs generated from hypoimmunogenic iPSCs were similar in cellular, molecular and functional characteristic as hRGCs generated from either hES or regular hiPS cells. They expressed guidance receptor genes for chemotropic cues recognition. When ROBO2 neutralized hiPSC/hES-RGCs were transplanted on chondroitinase treated retinal explants their incorporation within the RGC layer and beyond was better, compared to controls. Similarly, ROBO2 neutralized hiPSC-RGCs transplanted in chondroitinase treated eyes survived without an immunosuppressant and incorporated in the RGC layer better than controls.

Conclusions : The chemotropic environment may regulate the incorporation of transplanted hRGCs beside regulating axon pathfinding. The hypoimmunogenic hRGCs could evade immune surveillance and survive in the host retina suggesting the possibility of using off-the-shelf hypoimmunogenic iPSC lines for ex-vivo stem cell therapy for glaucoma.

This abstract was presented at the 2024 ARVO Annual Meeting, held in Seattle, WA, May 5-9, 2024.

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