Investigative Ophthalmology & Visual Science Cover Image for Volume 65, Issue 7
June 2024
Volume 65, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2024
Interrogating Temporality and Mechanisms of Retinogenesis in Human Retinal Organoids
Author Affiliations & Notes
  • Benvindo Chicha
    Biology, Johns Hopkins University, Baltimore, Maryland, United States
  • Robert Johnston
    Biology, Johns Hopkins University, Baltimore, Maryland, United States
  • Footnotes
    Commercial Relationships   Benvindo Chicha None; Robert Johnston None
  • Footnotes
    Support  None
Investigative Ophthalmology & Visual Science June 2024, Vol.65, 6686. doi:
  • Views
  • Share
  • Tools
    • Alerts
      ×
      This feature is available to authenticated users only.
      Sign In or Create an Account ×
    • Get Citation

      Benvindo Chicha, Robert Johnston; Interrogating Temporality and Mechanisms of Retinogenesis in Human Retinal Organoids. Invest. Ophthalmol. Vis. Sci. 2024;65(7):6686.

      Download citation file:


      © ARVO (1962-2015); The Authors (2016-present)

      ×
  • Supplements
Abstract

Purpose : The numerous neuronal types of the retina are generated at different overlapping time points, yet a high resolution timeline of cell birth, maturation, and death in the developing human retina has not been achieved, due to the experimental inaccessibility of human fetal retinal tissue. Moreover, the spatiotemporal mechanisms controlling neuronal development in the human retina are poorly understood. It is unclear if the temporality of cell fate specification reflects the birth timing or maturation rates of these cells. The main goal of this study is to identify temporal mechanisms governing the generation and maturation of neurons in the human retina using human retinal organoids.

Methods : To assess the timing of neuronal type birth, maturation, and death, I am using nucleotide analogs (i.e. EdU) to label newly replicated DNA for 48 hours every 7-days across organoid development. Whereas dividing progenitor cells will dilute the label over time, post-mitotic cells will retain this label, enabling me to track cells born at distinct times. The Johnston lab found that retinoic acid (RA) acts early and thyroid hormone (TH) acts late during retinogenesis to specify photoreceptor fates. To test how these signals affect the timing of cell generation, I am examining changes in cell birth dates upon changes in signaling environments.

Results : Preliminary experiments showed that EdU is toxic when administered in >48-hour pulses, limiting the length of the temporal windows. In a parallel approach, I am developing a transgene-based strategy to birth date cells with H2B-GFP in wider windows. Mitotic cells will divide and dilute the label while neurons born in the window will retain the H2B-GFP. This system allows more fine-tuned control and adaptability for later experiments.

Conclusions : This project will generate a timeline of human retinal neuron development at an unprecedented temporal and cell-type resolution, characterize new neuronal subtypes, and determine how signals affect the timing of their development. In addition, these studies will determine how signaling pathways govern the timing of cell type generation and cell fate specification decisions.

This abstract was presented at the 2024 ARVO Annual Meeting, held in Seattle, WA, May 5-9, 2024.

×
×

This PDF is available to Subscribers Only

Sign in or purchase a subscription to access this content. ×

You must be signed into an individual account to use this feature.

×