Investigative Ophthalmology & Visual Science Cover Image for Volume 65, Issue 7
June 2024
Volume 65, Issue 7
Open Access
ARVO Annual Meeting Abstract  |   June 2024
Regulation of Anti-VEGF Drug Effectiveness by Inflammatory Mediators in Patients with Diabetic Macular Edema
Author Affiliations & Notes
  • Susanne Mohr
    Physiology, Michigan State University, East Lansing, Michigan, United States
  • Sydney M Dankert
    Physiology, Michigan State University, East Lansing, Michigan, United States
  • Finn T Pötters
    Physiology, Michigan State University, East Lansing, Michigan, United States
  • Lucy S Candeub
    Physiology, Michigan State University, East Lansing, Michigan, United States
  • Louis C Glazer
    Vitreo-Retinal Associates PC, Grand Rapids, Michigan, United States
  • Brett Thomas Trombley
    Physiology, Michigan State University, East Lansing, Michigan, United States
  • Footnotes
    Commercial Relationships   Susanne Mohr GenenTech, Code F (Financial Support); Sydney Dankert GenenTech, Code F (Financial Support); Finn Pötters GenenTech, Code F (Financial Support); Lucy Candeub GenenTech, Code F (Financial Support); Louis Glazer GenenTech, Code F (Financial Support); Brett Trombley GeneTech, Code F (Financial Support)
  • Footnotes
    Support  GenenTech, ADA11-22-ICTSPM-15
Investigative Ophthalmology & Visual Science June 2024, Vol.65, 6261. doi:
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      Susanne Mohr, Sydney M Dankert, Finn T Pötters, Lucy S Candeub, Louis C Glazer, Brett Thomas Trombley; Regulation of Anti-VEGF Drug Effectiveness by Inflammatory Mediators in Patients with Diabetic Macular Edema. Invest. Ophthalmol. Vis. Sci. 2024;65(7):6261.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Purpose : Anti-VEGF (Vascular Endothelial Cell Growth Factor) drugs are the standard care for treatment of patients with diabetic macular edema (DME). Despite the success of anti-VEGF therapy roughly 30% of patients with DME do not respond properly to this type of therapeutic approach. Thus, there is a pressing need to understand whether and which retinal factors influence the effectiveness of anti-VEGF drugs, the aim of this project.

Methods : Diabetic patients (n=10) aged >18 years with central diabetic macular edema, BCVA >24 and <78, and central macular thickness (CMT) greater than 250 mm were enrolled in this study (ML39638). Following a full eye exam, imaging, and an aqueous tab, patients received ranibizumab (0.3mg/0.05mL) injections at day one and weeks four and eight. At week 12, a full eye exam, imaging, and a second aqueous tab was obtained prior to the last injection of ranibizumab. Pre- and post-treatment aqueous humor samples were then analyzed using Milliplex MAP magnetic bead assays.

Results : As expected, ranibizumab lowered levels of VEGF-A (p=0.0200), decreased CMT (central macular thickness, p=0.0480), and improved VA (visual acuity, p=0.0190). Interestingly, data analysis showed that the drug either had an immediate effect on vision parameters within 4 weeks after the first injection or none at all. Several pro-inflammatory mediators seemed to influence ranibizumab’s effectiveness in improving CMT. For example, baseline levels of interferon-γ correlated significantly with reduction in CMT (r=0.6490, p=0.0423). Other pro-inflammatory mediators such as the soluble receptors of IL-1 (interleukin-1) IL-1R1 (r=0.5985), IL-1R2 (r=0.6672), or Rantes (r=0.5640) had similar influence on ranibizumab’s effectiveness on lowering CMT.

Conclusions : Ranibizumab’s effectiveness can be determined within the first four weeks of therapy. Its effectiveness is greatly influenced by baseline levels of specific pro-inflammatory mediators. Fully identifying those pro-inflammatory mediators and their critical baseline levels will help to predict outcome of anti-VEGF therapies early in the treatment of diabetic macular edema.

This abstract was presented at the 2024 ARVO Annual Meeting, held in Seattle, WA, May 5-9, 2024.

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