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ARVO Annual Meeting Abstract June 2025

Modifying the VP1u Domain of AAV Improves Transduction of Murine, Canine, and Non-Human Primate Retinas by Intravitreal Injection

Mengtian Cui; Shun-Yun cheng; Bo Tian; Christine Harman; Amanda Jacobson; Lydia E Kapeller; Melaney A Mayes; Jackson McGowan; Haijiang Lin; Allison M Keeler; Andras M Komaromy; Meredith S Gregory-Ksander; Ann Marshak-Rothstein; Claudio Punzo; Phillip Tai; Guangping Gao

Invest. Ophthalmol. Vis. Sci.. 2025; 66(8):4652.

ARVO Annual Meeting Abstract June 2025

Microglia-derived Fas ligand drives axon degeneration and RGC death in the microbead-induced mouse model of Glaucoma

Meredith S Gregory-Ksander; Bhupender Verma; Cindy Hoppe; Sarah Dickman; Shintaro Shirahama; Drenushe Krasniqi; Ann Marshak-Rothstein; Bruce Ksander

Invest. Ophthalmol. Vis. Sci.. 2025; 66(8):2660.

ARVO Annual Meeting Abstract June 2024

Treatment with soluble Fas ligand prevents disease progression in the DBA/2J chronic mouse model of glaucoma

Sarah Dickman; Anitha Krishnan; Bhupender Verma; Ann Marshak-Rothstein; Meredith S Gregory-Ksander

Invest. Ophthalmol. Vis. Sci.. 2024; 65(7):946.

ARVO Annual Meeting Abstract June 2022

Uncoupling caspase 8-mediated-apotosis from caspase 8-mediated-inflammation in glaucoma

Yinjie Guo; Maleeka Shrestha; Ann Marshak-Rothstein; Meredith S Gregory-Ksander

Invest. Ophthalmol. Vis. Sci.. 2022; 63(7):1610 – A0433.

ARVO Annual Meeting Abstract June 2022

Overexpression of membrane bound Fas ligand accelerates inflammation in response to corneal alkali burns.

Sean Doherty; Wenqi Su; Shuo Sun; Evan Bilsbury; Emma Wood; Haijiang Lin; Ann Marshak-Rothstein; Bo Tian

Invest. Ophthalmol. Vis. Sci.. 2022; 63(7):3236 – A0271.

ARVO Annual Meeting Abstract June 2021

Caspase-8-mediated inflammation but not apoptosis drives the development of glaucoma

Yinjie Guo; Maleeka Shrestha; Ann Marshak-Rothstein; Meredith S Gregory-Ksander

Invest. Ophthalmol. Vis. Sci.. 2021; 62(8):2350.

ARVO Annual Meeting Abstract June 2020

Intravitreal administration of AAV2.soluble Fas ligand gene therapy following glaucoma injury restores retinal ganglion cell function and stops disease progression

Meredith S Gregory-Ksander; Maleeka Shrestha; Khoa Nguyen; Ann Marshak-Rothstein; Anitha Krishnan

Invest. Ophthalmol. Vis. Sci.. 2020; 61(7):2366.

ARVO Annual Meeting Abstract June 2017

Destructive neuroinflammation triggered by activation of the NLRP3 inflammasome in the glaucomatous optic nerve head

Meredith S Gregory-Ksander; Fei Fei; anitha krishnan; Te-Chen Tzeng; Deepa Subramanian; Douglas Golenbock; Ann Marshak-Rothstein; Bruce Ksander

Invest. Ophthalmol. Vis. Sci.. 2017; 58(8):2021.

ARVO Annual Meeting Abstract September 2016

Soluble Fas ligand provides long-term protection in a chronic mouse model of glaucoma by inhibiting glial activation, inflammation, and apoptosis

Meredith S Gregory-Ksander; Fei Fei; Ann Marshak-Rothstein; Bruce Ksander; Anitha Krishnan

Invest. Ophthalmol. Vis. Sci.. 2016; 57(12)

ARVO Annual Meeting Abstract June 2015

Preventing glaucoma by blocking activation of the NLRP3 inflammasome in the optic nerve head

Fei Fei; Anitha Krishnan; Ann Marshak Rothstein; Bruce R Ksander; Meredith S Gregory-Ksander

Invest. Ophthalmol. Vis. Sci.. 2015; 56(7 ):3485.

ARVO Annual Meeting Abstract June 2015

Vascular normalization: A novel method for inhibiting vascular leakage in age-related macular degeneration

Adarsha Koirala; Ann Marshak Rothstein; Bruce R Ksander; Meredith S Gregory-Ksander

Invest. Ophthalmol. Vis. Sci.. 2015; 56(7 ):4800.

ARVO Annual Meeting Abstract June 2015

Gene therapy treatment with AAV-soluble Fas ligand protects retinal ganglion cells during development of Glaucoma

Anitha Krishnan; Fei Fei; Alexander Jones; Bruce R Ksander; Ann Marshak Rothstein; Meredith S Gregory-Ksander

Invest. Ophthalmol. Vis. Sci.. 2015; 56(7 ):2594.

ARVO Annual Meeting Abstract April 2014

Soluble Fas ligand is a neuroprotective molecule critical in restricting glaucomatous damage to retinal ganglion cells

Anitha Krishnan; Alexander Jones; Ann Marshak Rothstein; Bruce R Ksander; Meredith S Gregory-Ksander

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):2194.

ARVO Annual Meeting Abstract April 2014

Regulation of vascular leakage by Fas Ligand: Distinguishing the role of soluble from membrane FasL in unique knock-in mice with laser-induced choroidal neovascularization

Adarsha Koirala; Ann Marshak-Rothstein; Bruce R Ksander; Meredith S Gregory-Ksander

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):80.

ARVO Annual Meeting Abstract April 2011

Cleavage of Fas Ligand Regulates Vessel Leakage in Laser Induced CNV

Carolin Milojcic; Dimosthenis Mantopoulos; Demetrios Vavvas; Andrea Giani; Ann Marshak-Rothstein; Frank G. Holz; Bruce R. Ksander; Meredith S. Gregory

Invest. Ophthalmol. Vis. Sci.. 2011; 52(14):2300.

ARVO Annual Meeting Abstract April 2011

Accelerated Glaucoma And Increased Neurotoxicity In DBA/2J Mice That Cannot Cleave Fas Ligand

Bruce R. Ksander; Alexander Jones; Simon W. John; Ann Marshak-Rothstein; Meredith S. Gregory

Invest. Ophthalmol. Vis. Sci.. 2011; 52(14):2449.

ARVO Annual Meeting Abstract April 2010

Microglia Are Neurotoxic or Neuroprotective During the Development of Glaucoma Depending Upon the Expression of Membrane and Soluble FasL

M. S. Gregory; C. G. Hackett; E. Abernathy; A. Jones; M. R. Crane; A. Marshak-Rothstein; K. Lee; H. Chen; D. Chen; B. R. Ksander

Invest. Ophthalmol. Vis. Sci.. 2010; 51(13):3190.

ARVO Annual Meeting Abstract April 2009

Both Membrane And Soluble Fas Ligand Are Required For Clearance Of Bacteria And Protection Of Host Tissue During S. aureus Induced Endophthalmitis.

C. G. Hackett; A. Marshak-Rothstein; M. S. Gregory

Invest. Ophthalmol. Vis. Sci.. 2009; 50(13):851.

ARVO Annual Meeting Abstract April 2009

The Form of Fas Ligand Determines Whether Retinal Ganglion Cells Survive or Die During Glaucoma

B. R. Ksander; C. G. Hackett; E. F. Abernathy; A. Marshak-Rothstein; K. Lee; D. F. Chen; M. S. Gregory

Invest. Ophthalmol. Vis. Sci.. 2009; 50(13):5925.

ARVO Annual Meeting Abstract May 2007

Age-Related Spontaneous Corneal Lesions That Are Found Predominantly in Female CD36KO Mice

R. N. Barcia; S. Masli; K. Lee; A. Marshak-Rothstein; J. Zieske; M. Gregory-Ksander; B. R. Ksander

Invest. Ophthalmol. Vis. Sci.. 2007; 48(13):4324.

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Modifying the VP1u Domain of AAV Improves Transduction of Murine, Canine, and Non-Human Primate Retinas by Intravitreal Injection

Microglia-derived Fas ligand drives axon degeneration and RGC death in the microbead-induced mouse model of Glaucoma

Treatment with soluble Fas ligand prevents disease progression in the DBA/2J chronic mouse model of glaucoma

Uncoupling caspase 8-mediated-apotosis from caspase 8-mediated-inflammation in glaucoma

Overexpression of membrane bound Fas ligand accelerates inflammation in response to corneal alkali burns.

Caspase-8-mediated inflammation but not apoptosis drives the development of glaucoma

Intravitreal administration of AAV2.soluble Fas ligand gene therapy following glaucoma injury restores retinal ganglion cell function and stops disease progression

Destructive neuroinflammation triggered by activation of the NLRP3 inflammasome in the glaucomatous optic nerve head

Soluble Fas ligand provides long-term protection in a chronic mouse model of glaucoma by inhibiting glial activation, inflammation, and apoptosis

Preventing glaucoma by blocking activation of the NLRP3 inflammasome in the optic nerve head

Vascular normalization: A novel method for inhibiting vascular leakage in age-related macular degeneration

Gene therapy treatment with AAV-soluble Fas ligand protects retinal ganglion cells during development of Glaucoma

Soluble Fas ligand is a neuroprotective molecule critical in restricting glaucomatous damage to retinal ganglion cells

Regulation of vascular leakage by Fas Ligand: Distinguishing the role of soluble from membrane FasL in unique knock-in mice with laser-induced choroidal neovascularization

Cleavage of Fas Ligand Regulates Vessel Leakage in Laser Induced CNV

Accelerated Glaucoma And Increased Neurotoxicity In DBA/2J Mice That Cannot Cleave Fas Ligand

Microglia Are Neurotoxic or Neuroprotective During the Development of Glaucoma Depending Upon the Expression of Membrane and Soluble FasL

Both Membrane And Soluble Fas Ligand Are Required For Clearance Of Bacteria And Protection Of Host Tissue During S. aureus Induced Endophthalmitis.

The Form of Fas Ligand Determines Whether Retinal Ganglion Cells Survive or Die During Glaucoma

Age-Related Spontaneous Corneal Lesions That Are Found Predominantly in Female CD36KO Mice

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