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Immunology and Microbiology November 2013

ITE, A Novel Endogenous Nontoxic Aryl Hydrocarbon Receptor Ligand, Efficiently Suppresses EAU and T-Cell–Mediated Immunity

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Lindsey F. Nugent; Guangpu Shi; Barbara P. Vistica; Osato Ogbeifun; Samuel J. H. Hinshaw; Igal Gery
TAGS: aryl hydrocarbon receptor, cytokine, immune response, cell-mediated, interleukin-17, ligands, mice, foxp3 gene, antibodies, immune response, interleukin-10
Invest. Ophthalmol. Vis. Sci.. 2013; 54(12):7463-7469. https://doi.org/10.1167/iovs.12-11479

ARVO Annual Meeting Abstract March 2012

Cell-cell Interaction with APC, not IL-23, is Required for Naive CD4 Cells to Acquire Pathogenicity During Th17 Lineage Commitment

Guangpu Shi; Jenna D. Lovaas; Cuiyan Tan; Barbara P. Vistica; Eric F. Wawrousek; Mehak K. Aziz; Rachael C. Rigden; Rachel R. Caspi; Igal Gery

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):5487.

ARVO Annual Meeting Abstract March 2012

DAP-12, a Major Immunomediator, Either Promotes or Suppresses EAU Development

Barbara P. Vistica; Vanessa Montalvo-Reddin; Guangpu Shi; Lindsey Nugent; Laura Quigley; Daniel W. McVicar; Igal Gery

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):6239.

ARVO Annual Meeting Abstract March 2012

A Novel Nontoxic Aryl Hydrocarbon Receptor Ligand, ITE, Suppresses Immune-mediated Ocular Inflammation

Lindsey F. Nugent; Guangpu Shi; Barbara P. Vistica; Cuiyan Tan; Igal Gery

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):2759.

ARVO Annual Meeting Abstract April 2011

SLAT/Def6 Knock-Out (KO) Mice Exhibit Profoundly Reduced Capacity to Develop Experimental Autoimmune Uveitis (EAU)

Barbara P. Vistica; Guangpu Shi; Lindsey Nugent; Amnon Altman; Igal Gery

Invest. Ophthalmol. Vis. Sci.. 2011; 52(14):2926.

ARVO Annual Meeting Abstract April 2011

Antigen-Specific Th9 Cells are Unique in their Cytokine Kinetics, Plasticity and Retention at the Inflammatory Site

Lindsey F. Nugent; Cuiyan Tan; Mehak Aziz; Barbara P. Vistica; Guangpu Shi; Igal Gery

Invest. Ophthalmol. Vis. Sci.. 2011; 52(14):3011.

ARVO Annual Meeting Abstract April 2011

Acquisition of Th1, Th17, or Treg Phenotypes Differs Among CD4 T Cells in Ocular Inflammation Triggered by Different TLR Ligands

Guangpu Shi; Barbara P. Vistica; Lindsey F. Nugent; Eric F. Wawrousek; Igal Gery

Invest. Ophthalmol. Vis. Sci.. 2011; 52(14):2279.

ARVO Annual Meeting Abstract April 2010

Interferon Beta Down-Regulates Cxcr3 on T Lymphocytes and Prevents Mice From Developing Immune-Mediated Ocular Inflammation

J. Chen; C. Tan; B. P. Vistica; I. Gery; B. Detrick; J. J. Hooks

Invest. Ophthalmol. Vis. Sci.. 2010; 51(13):818.

ARVO Annual Meeting Abstract April 2010

A Comparison of the Biological Activities of the Pathogenic and Non-Pathogenic Subsets of the Th17 Cells

B. P. Vistica; G. Shi; J. Lovaas; C. Tan; M. Aziz; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2010; 51(13):822.

ARVO Annual Meeting Abstract April 2010

Generation, Unique Features and Pathogenicity of T-Helper Cells Selectively Producing Il-9 ("th9")

M. Aziz; C. Tan; B. P. Vistica; G. Shi; J. D. Lovaas; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2010; 51(13):2017.

ARVO Annual Meeting Abstract April 2009

Expression Profiles of Apoptosis-Related and Co-Inhibitory Molecules by Eyes Developing Th1- or Th17-Induced Inflammation

C. Tan; G. Shi; B. P. Vistica; E. F. Wawrousek; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2009; 50(13):5548.

ARVO Annual Meeting Abstract April 2009

Th17 Cells, but Not Th1, Mediate Sustained Autoimmune Inflammation in the Eye by Their Relative Resistance to Restimulation-Induced Cell Death (RICD)

B. P. Vistica; G. Shi; M. Ramaswamy; C. Tan; E. F. Wawrousek; R. M. Siegel; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2009; 50(13):2633.

ARVO Annual Meeting Abstract April 2009

A Subpopulation of Th17 Cells Produce High Levels of IL-17, but Fail to Adoptively Transfer Ocular Inflammation

J. D. Lovaas; G. Shi; C. Tan; B. P. Vistica; E. F. Wawrousek; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2009; 50(13):2640.

ARVO Annual Meeting Abstract May 2008

Phenotype Switching by Inflammation-Inducing Polarized T-Helper (Th)17 Cells, but Not by Th1 Cells

G. Shi; C. A. Cox; B. P. Vistica; E. F. Wawrousek; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2008; 49(13):1991.

ARVO Annual Meeting Abstract May 2008

Selective Polarization Toward Th17 or Th1 Phenotypes in Mouse Eyes With Inflammation Due to Local Expression of IL-1 or IL-7

C. Tan; G. Shi; B. P. Vistica; E. F. Wawrousek; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2008; 49(13):2529.

ARVO Annual Meeting Abstract May 2008

Microarray Analysis of Gene Expression in Light Exposed Mouse Retina: A Comparison Between Light-Damage Susceptible and Resistant Strains

H. Takase; C. C. Chan; M. Santos-Muffley; J. Tuo; B. P. Vistica; M. Mochizuki; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2008; 49(13):4417.

ARVO Annual Meeting Abstract May 2008

Comparison of Amino Acid Copolymers Glatiramer Acetate (GA) and poly(Y, F, A, K)n in Inhibition of Development of Experimental Autoimmune Uveitis (EAU) and Related Immune Responses

B. P. Vistica; H. Yin; C.-C. Chan; J. L. Strominger; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2008; 49(13):4764.

ARVO Annual Meeting Abstract May 2007

Homeostatic Activation Triggers Pathogenicity in Naive T-Cells

B. P. Vistica; A. B. D. Collins; H. Yin; C. Fujimoto; S. J. Kim; E. F. Wawrousek; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2007; 48(13):2644.

ARVO Annual Meeting Abstract May 2007

Pertussis Toxin (PTX) is Superior to Other Toll-Like Receptor (TLR) Ligands in Shifting T-helper (Th) Cell Populations Towards Th17

G. Shi; B. P. Vistica; C.-R. Yu; C. A. Cox; V. Montalvo; E. F. Wawrousek; C. E. Egwuagu; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2007; 48(13):2832.

ARVO Annual Meeting Abstract May 2007

Polarized TCR-Transgenic Th17 Cells Resemble Th1 Cells in Their Capacity to Adoptively Transfer Ocular Inflammation, but Differ in Other Biological Activities

C. A. Cox; G. Shi; H. Yin; B. P. Vistica; E. F. Wawrousek; C.-C. Chan; I. Gery

Invest. Ophthalmol. Vis. Sci.. 2007; 48(13):2834.

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ITE, A Novel Endogenous Nontoxic Aryl Hydrocarbon Receptor Ligand, Efficiently Suppresses EAU and T-Cell–Mediated Immunity

Cell-cell Interaction with APC, not IL-23, is Required for Naive CD4 Cells to Acquire Pathogenicity During Th17 Lineage Commitment

DAP-12, a Major Immunomediator, Either Promotes or Suppresses EAU Development

A Novel Nontoxic Aryl Hydrocarbon Receptor Ligand, ITE, Suppresses Immune-mediated Ocular Inflammation

SLAT/Def6 Knock-Out (KO) Mice Exhibit Profoundly Reduced Capacity to Develop Experimental Autoimmune Uveitis (EAU)

Antigen-Specific Th9 Cells are Unique in their Cytokine Kinetics, Plasticity and Retention at the Inflammatory Site

Acquisition of Th1, Th17, or Treg Phenotypes Differs Among CD4 T Cells in Ocular Inflammation Triggered by Different TLR Ligands

Interferon Beta Down-Regulates Cxcr3 on T Lymphocytes and Prevents Mice From Developing Immune-Mediated Ocular Inflammation

A Comparison of the Biological Activities of the Pathogenic and Non-Pathogenic Subsets of the Th17 Cells

Generation, Unique Features and Pathogenicity of T-Helper Cells Selectively Producing Il-9 ("th9")

Expression Profiles of Apoptosis-Related and Co-Inhibitory Molecules by Eyes Developing Th1- or Th17-Induced Inflammation

Th17 Cells, but Not Th1, Mediate Sustained Autoimmune Inflammation in the Eye by Their Relative Resistance to Restimulation-Induced Cell Death (RICD)

A Subpopulation of Th17 Cells Produce High Levels of IL-17, but Fail to Adoptively Transfer Ocular Inflammation

Phenotype Switching by Inflammation-Inducing Polarized T-Helper (Th)17 Cells, but Not by Th1 Cells

Selective Polarization Toward Th17 or Th1 Phenotypes in Mouse Eyes With Inflammation Due to Local Expression of IL-1 or IL-7

Microarray Analysis of Gene Expression in Light Exposed Mouse Retina: A Comparison Between Light-Damage Susceptible and Resistant Strains

Comparison of Amino Acid Copolymers Glatiramer Acetate (GA) and poly(Y, F, A, K)n in Inhibition of Development of Experimental Autoimmune Uveitis (EAU) and Related Immune Responses

Homeostatic Activation Triggers Pathogenicity in Naive T-Cells

Pertussis Toxin (PTX) is Superior to Other Toll-Like Receptor (TLR) Ligands in Shifting T-helper (Th) Cell Populations Towards Th17

Polarized TCR-Transgenic Th17 Cells Resemble Th1 Cells in Their Capacity to Adoptively Transfer Ocular Inflammation, but Differ in Other Biological Activities

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