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ARVO Annual Meeting Abstract June 2025

D1 and D4 Antagonist effects on light adaptation using ex-vivo ERGs after 6 weeks of diabetes

David Wayne Umbertus; Angela MacIsaac; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2025; 66(8):5590.

ARVO Annual Meeting Abstract June 2025

Calcium Handling For Optogenetically Elicited Inhibition Onto Diabetic Rod Bipolar Cells

Jared Hill; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2025; 66(8):2314.

ARVO Annual Meeting Abstract June 2024

Light adaptation of ex-vivo ERGs is limited after 6 weeks of diabetes

Angela MacIsaac; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2024; 65(7):3928.

ARVO Annual Meeting Abstract June 2024

Optogenetically Elicited Inhibition On Rod Bipolar Cells and On Cone Bipolar Cells

Jared Hill; Timothy David Maley; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2024; 65(7):3921.

ARVO Annual Meeting Abstract June 2024

DOPAMINE 1 RECEPTOR ACTIVATION MODULATES OPTOGENETIC ELICITED GLYCINERGIC BUT NOT GABAergic INHIBITION ON OFF BIPOLAR CELLS

Timothy David Maley; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2024; 65(7):5360.

ARVO Annual Meeting Abstract June 2023

D1R AND D4R AGONISTS MODIFY LIGHT-EVOKED WHOLE-RETINA RECORDINGS

Angela MacIsaac; Kyle Filicetti; Andrea Wellington; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2023; 64(8):42.

ARVO Annual Meeting Abstract June 2023

OPTOGENETIC ELICITED INHIBITION ON AMACRINE OR OFF BIPOLAR CELLS IS NOT MODULATED BY DOPAMINE 1 RECEPTOR ACTIVATION.

Timothy Maley; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2023; 64(8):1662.

Visual Neuroscience January 2022

Impaired Light Adaptation of ON-Sustained Ganglion Cells in Early Diabetes Is Attributable to Diminished Response to Dopamine D4 Receptor Activation

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Michael D. Flood; Andrea J. Wellington; Erika D. Eggers
TAGS: diabetes mellitus, ganglia, light adaptation, dopamine d4 receptor, dopamine receptors
Invest. Ophthalmol. Vis. Sci.. 2022; 63(1):33. https://doi.org/10.1167/iovs.63.1.33

ARVO Annual Meeting Abstract June 2020

Impaired light adaptation in early diabetes is not accompanied by major cell death or dopamine receptor insensitivity

Michael Flood; Erika D Eggers; Andrea Wellington; Luis Cruz

Invest. Ophthalmol. Vis. Sci.. 2020; 61(7):2729.

ARVO Annual Meeting Abstract June 2020

The Use of Optogenetics to Study Inhibition in the Ex Vivo Mouse Electroretinogram

Timothy Maley; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2020; 61(7):5058.

Visual Neuroscience September 2019

Reductions in Calcium Signaling Limit Inhibition to Diabetic Retinal Rod Bipolar Cells

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Johnnie M. Moore-Dotson; Erika D. Eggers
TAGS: amacrine cells, diabetes mellitus, gamma-aminobutyric acid, retinal bipolar cells, rod photoreceptors, diabetes mellitus, type 2
Invest. Ophthalmol. Vis. Sci.. 2019; 60(12):4063-4073. https://doi.org/10.1167/iovs.19-27137

ARVO Annual Meeting Abstract July 2019

Dopamine decreases excitatory inputs to ON sustained ganglion cells via both D1 and D4 receptor-dependent pathways

Michael Flood; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2019; 60(9):557.

ARVO Annual Meeting Abstract June 2017

Inhibition in the retinal ON cone pathway is increased in early diabetes

Johnnie Moore-Dotson; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2017; 58(8):1220.

ARVO Annual Meeting Abstract June 2017

Light adaptation is impaired at the ganglion cell level after six weeks of diabetes

Michael Flood; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2017; 58(8):5857.

ARVO Annual Meeting Abstract September 2016

Dopamine D1 receptors are involved in light-adapted changes to bipolar cell spontaneous inhibition and receptive field surrounds

Erika D Eggers; Reece Mazade; Michael Flood

Invest. Ophthalmol. Vis. Sci.. 2016; 57(12):594.

ARVO Annual Meeting Abstract September 2016

Light-evoked GABA release and A17 amacrine cell reciprocal inhibition to rod bipolar cells are decreased in a mouse model of diabetes

Johnnie Moore-Dotson; Reece Mazade; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2016; 57(12)

Visual Neuroscience March 2016

Early Retinal Neuronal Dysfunction in Diabetic Mice: Reduced Light-Evoked Inhibition Increases Rod Pathway Signaling

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Johnnie M. Moore-Dotson; Jamie J. Beckman; Reece E. Mazade; Mrinalini Hoon; Adam S. Bernstein; Melissa J. Romero-Aleshire; Heddwen L. Brooks; Erika D. Eggers
TAGS: diabetes mellitus, retinal bipolar cells, rod photoreceptors, diabetes mellitus, type 2, mice, gamma-aminobutyric acid, amacrine cells, signal transduction
Invest. Ophthalmol. Vis. Sci.. 2016; 57(3):1418-1430. https://doi.org/10.1167/iovs.15-17999

ARVO Annual Meeting Abstract June 2015

Light adaptation narrows spatial inhibitory input and increases signal output of the mouse inner retina

Reece Mazade; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2015; 56(7 ):3223.

ARVO Annual Meeting Abstract June 2015

Amacrine cell dysfunction in early diabetes

Johnnie Moore-Dotson; Reece Mazade; Adam Bernstein; Melissa Romero-Aleshire; Heddwen Brooks; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2015; 56(7 ):3229.

ARVO Annual Meeting Abstract April 2014

Spatial inhibitory input to the retinal OFF pathway narrows with light adaptation

Reece Mazade; Erika D Eggers

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):5007.

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D1 and D4 Antagonist effects on light adaptation using ex-vivo ERGs after 6 weeks of diabetes

Calcium Handling For Optogenetically Elicited Inhibition Onto Diabetic Rod Bipolar Cells

Light adaptation of ex-vivo ERGs is limited after 6 weeks of diabetes

Optogenetically Elicited Inhibition On Rod Bipolar Cells and On Cone Bipolar Cells

DOPAMINE 1 RECEPTOR ACTIVATION MODULATES OPTOGENETIC ELICITED GLYCINERGIC BUT NOT GABAergic INHIBITION ON OFF BIPOLAR CELLS

D1R AND D4R AGONISTS MODIFY LIGHT-EVOKED WHOLE-RETINA RECORDINGS

OPTOGENETIC ELICITED INHIBITION ON AMACRINE OR OFF BIPOLAR CELLS IS NOT MODULATED BY DOPAMINE 1 RECEPTOR ACTIVATION.

Impaired Light Adaptation of ON-Sustained Ganglion Cells in Early Diabetes Is Attributable to Diminished Response to Dopamine D4 Receptor Activation

Impaired light adaptation in early diabetes is not accompanied by major cell death or dopamine receptor insensitivity

The Use of Optogenetics to Study Inhibition in the Ex Vivo Mouse Electroretinogram

Reductions in Calcium Signaling Limit Inhibition to Diabetic Retinal Rod Bipolar Cells

Dopamine decreases excitatory inputs to ON sustained ganglion cells via both D1 and D4 receptor-dependent pathways

Inhibition in the retinal ON cone pathway is increased in early diabetes

Light adaptation is impaired at the ganglion cell level after six weeks of diabetes

Dopamine D1 receptors are involved in light-adapted changes to bipolar cell spontaneous inhibition and receptive field surrounds

Light-evoked GABA release and A17 amacrine cell reciprocal inhibition to rod bipolar cells are decreased in a mouse model of diabetes

Early Retinal Neuronal Dysfunction in Diabetic Mice: Reduced Light-Evoked Inhibition Increases Rod Pathway Signaling

Light adaptation narrows spatial inhibitory input and increases signal output of the mouse inner retina

Amacrine cell dysfunction in early diabetes

Spatial inhibitory input to the retinal OFF pathway narrows with light adaptation

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