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ARVO Annual Meeting Abstract June 2021

Pegylated Arginase 1 as a Treatment for Acute Central Nervous System (CNS) Injury

Abdelrahman Fouda; Wael Eldahshan; Zhimin Xu; Tahira Lemtalsi; Esraa Shosha; Paul Ning-Man Cheng; S. Priya Narayanan; Robert W Caldwell; Ruth B Caldwell

Invest. Ophthalmol. Vis. Sci.. 2021; 62(8):2999.

ARVO Annual Meeting Abstract June 2021

Arginase Isoforms and Diabetic Retinopathy in the db/db Mouse

Katharine Leilani Bunch; Ruth B Caldwell; Robert W Caldwell

Invest. Ophthalmol. Vis. Sci.. 2021; 62(8):3031.

ARVO Annual Meeting Abstract June 2021

Novel role of acyl-coenzyme A: cholesterol transferase (ACAT1) in ischemic retinopathy

Modesto Antonio Rojas; Tahira Lemtalsi; Isabella Santana; Robert W Caldwell; Ruth B Caldwell

Invest. Ophthalmol. Vis. Sci.. 2021; 62(8):3015.

ARVO Annual Meeting Abstract June 2021

Role for Arg1 and Arg2 in Age-related Intraocular Pressure Homeostasis

Jingwen Cai; Hongfang Yu; Michelle Drewry; Daniel W Stamer; Robert W Caldwell; Ruth B Caldwell; Yutao Liu

Invest. Ophthalmol. Vis. Sci.. 2021; 62(8):1492.

ARVO Annual Meeting Abstract June 2020

Development of pegylated arginase 1 as a therapy for retinal ischemic injury

Abdelrahman Y. Fouda; Zhimin Xu; Esraa Shosha; Paul Ning-Man Cheng; S. Priya Narayanan; Robert W Caldwell; Ruth B Caldwell

Invest. Ophthalmol. Vis. Sci.. 2020; 61(7):4052.

ARVO Annual Meeting Abstract July 2019

Targeting neurovascular injury: Arginase 1 promotes angiogenic repair and limits neurodegeneration

Ruth B Caldwell; Abdelrahman Y Fouda; Zhimin Xu; Jutamas Suwanpradid; Modesto Rojas; Esraa Shosha; Chintan Patel; Ji Xing; S. Priya Narayanan; Robert W. Caldwell

Invest. Ophthalmol. Vis. Sci.. 2019; 60(9):3258.

ARVO Annual Meeting Abstract April 2014

Inhibition of polyamine oxidase reduces hyperoxia-mediated retinal neuro-vascular injury in a model of retinopathy of prematurity

S. Priya Narayanan; Zhimin Xu; Tahira Lemtalsi; Robert William Caldwell; Ruth B Caldwell

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):1716.

ARVO Annual Meeting Abstract April 2014

Arginase 2 deletion limits hyperoxia-induced retinal vascular injury through normalization of NOS function and upregulation of arginase 1

Jutamas Suwanpradid; Modesto Antonio Rojas; Robert William Caldwell; Ruth B Caldwell

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):1739.

ARVO Annual Meeting Abstract April 2014

Arginase blockade preserves retinal neurons during ischemia/reperfusion injury

Zhimin Xu; Esraa Shosha; S. Priya Narayanan; Harumasa Yokota; Robert William Caldwell; Ruth B Caldwell

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):1898.

ARVO Annual Meeting Abstract April 2014

Endoplasmic Reticulum Stress-regulated CXCL10/CXCR3 pathway mediates inflammation and neuronal injury after retinal ischemia

Yonju Ha; Hua Liu; Zhimin Xu; Harumasa Yokota; Tahira Lemtalsi; Sylvia B Smith; Robert William Caldwell; Massoud Motamedi; Ruth B Caldwell; Wenbo Zhang

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):2178.

ARVO Annual Meeting Abstract April 2014

Mechanistic role of arginase in inducing endothelial cell senescence in diabetic retinopathy

Esraa Shosha; Tahira Lemtalsi; Zhimin Xu; Robert William Caldwell; Ruth B Caldwell; S. Priya Narayanan

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):3557.

ARVO Annual Meeting Abstract April 2014

Diabetes induced-injury in retina and bone marrow cells involves NOX2-induced activation of arginase

Modesto Antonio Rojas; S. Priya Narayanan; Haroldo A Toque; Tahira Lemtalsi; Zhimin Xu; David Fulton; Robert William Caldwell; Ruth B Caldwell

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):4905.

ARVO Annual Meeting Abstract April 2014

Endothelin, a novel angiogenic factor in ischemic retinopathy

Chintan Patel; Wenbo Zhang; S. Priya Narayanan; Zhimin Xu; Robert William Caldwell; Ruth B Caldwell

Invest. Ophthalmol. Vis. Sci.. 2014; 55(13):5392.

Retinal Cell Biology February 2013

Hyperoxia Causes Regression of Vitreous Neovascularization by Downregulating VEGF/VEGFR2 Pathway

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Hua Liu; Wenbo Zhang; Zhimin Xu; Robert W. Caldwell; Ruth B. Caldwell; Steven E. Brooks
TAGS: hyperoxia, neovascularization, pathologic, vascular endothelial growth factor a, vascular endothelial growth factor receptor-2, vitreous body, vitreous neovascularization, apoptosis, ischemia, retinal diseases, oxygen
Invest. Ophthalmol. Vis. Sci.. 2013; 54(2):918-931. https://doi.org/10.1167/iovs.12-11291

Includes: Supplemental Content

ARVO Annual Meeting Abstract March 2012

Deletion of Arginase 2 Prevents Retinal Ganglion Cell Loss and Blocks Formation of Acellular Capillaries after Ischemia/Reperfusion Injury

Zhimin Xu; Harumasa Yokota; Subhadra P. Narayanan; Robert W. Caldwell; Ruth B. Caldwell

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):5329.

ARVO Annual Meeting Abstract March 2012

Arginase 2 Deficiency Limits Microglia/Macrophage Activation and Prevents Hyperoxia-induced Vascular Injury in the Mouse Retina

Jutamas Suwanpradid; Zhimin Xu; Subhadra P. Narayanan; Robert W. Caldwell; Ruth B. Caldwell

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):5882.

ARVO Annual Meeting Abstract March 2012

Arginase2 Deficiency Reduces Hyperoxia-induced Retinal Neurodegeneration through the Regulation of Polyamine Metabolism

S. P. Narayanan; Jutamas Suwanpradid; Zhimin Xu; Tahira Lemtalsi; Nagireddy Putluri; Arun Sreekumar; Robert W. Caldwell; Ruth B. Caldwell

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):6418.

ARVO Annual Meeting Abstract March 2012

Deficiency of CXCR3 Prevents Inflammation and Neuronal Damage in Retinal Ischemic Injury

Wenbo Zhang; Hua Liu; Zhimin Xu; Harumasa Yokota; Jun Wang; Subhadra P. Narayanan; Modesto A. Rojas; Massoud Motamedi; Robert W. Caldwell; Ruth B. Caldwell

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):1672.

ARVO Annual Meeting Abstract March 2012

Mechanisms of Diabetic Retinopathy: Role of NOX2 in Retinal and Bone Marrow-derived Cells

Modesto A. Rojas; Wenbo Zhang; Zhimin Xu; Phillip Chandler; Robert W. Caldwell; Ruth B. Caldwell

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):2419.

ARVO Annual Meeting Abstract March 2012

Hyperoxia Causes Regression of Established Vitreous Neovascularization By Targeting the VEGF/VEGFR-2 Survival Pathway

Steven E. Brooks; Wenbo Zhang; Hua Liu; Zhimin Xu; Harumasa Yokota; Robert W. Caldwell; Akitoshi Yoshida; Ruth B. Caldwell

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):2531.

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Pegylated Arginase 1 as a Treatment for Acute Central Nervous System (CNS) Injury

Arginase Isoforms and Diabetic Retinopathy in the db/db Mouse

Novel role of acyl-coenzyme A: cholesterol transferase (ACAT1) in ischemic retinopathy

Role for Arg1 and Arg2 in Age-related Intraocular Pressure Homeostasis

Development of pegylated arginase 1 as a therapy for retinal ischemic injury

Targeting neurovascular injury: Arginase 1 promotes angiogenic repair and limits neurodegeneration

Inhibition of polyamine oxidase reduces hyperoxia-mediated retinal neuro-vascular injury in a model of retinopathy of prematurity

Arginase 2 deletion limits hyperoxia-induced retinal vascular injury through normalization of NOS function and upregulation of arginase 1

Arginase blockade preserves retinal neurons during ischemia/reperfusion injury

Endoplasmic Reticulum Stress-regulated CXCL10/CXCR3 pathway mediates inflammation and neuronal injury after retinal ischemia

Mechanistic role of arginase in inducing endothelial cell senescence in diabetic retinopathy

Diabetes induced-injury in retina and bone marrow cells involves NOX2-induced activation of arginase

Endothelin, a novel angiogenic factor in ischemic retinopathy

Hyperoxia Causes Regression of Vitreous Neovascularization by Downregulating VEGF/VEGFR2 Pathway

Deletion of Arginase 2 Prevents Retinal Ganglion Cell Loss and Blocks Formation of Acellular Capillaries after Ischemia/Reperfusion Injury

Arginase 2 Deficiency Limits Microglia/Macrophage Activation and Prevents Hyperoxia-induced Vascular Injury in the Mouse Retina

Arginase2 Deficiency Reduces Hyperoxia-induced Retinal Neurodegeneration through the Regulation of Polyamine Metabolism

Deficiency of CXCR3 Prevents Inflammation and Neuronal Damage in Retinal Ischemic Injury

Mechanisms of Diabetic Retinopathy: Role of NOX2 in Retinal and Bone Marrow-derived Cells

Hyperoxia Causes Regression of Established Vitreous Neovascularization By Targeting the VEGF/VEGFR-2 Survival Pathway

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