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Retinal Cell Biology September 2011

Reduced Retinal Neovascularization, Vascular Permeability, and Apoptosis in Ischemic Retinopathy in the Absence of Prolyl Hydroxylase-1 Due to the Prevention of Hyperoxia-Induced Vascular Obliteration

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Hu Huang; Sara Van de Veire; Mansi Dalal; Rachel Parlier; Richard D. Semba; Peter Carmeliet; Stanley A. Vinores
TAGS: apoptosis, hyperoxia, hypoxia, ischemia, mice, mixed function oxygenases, prevention, retinal diseases, retinal neovascularization, vascular endothelial growth factor a
Invest. Ophthalmol. Vis. Sci.. 2011; 52(10):7565-7573. https://doi.org/10.1167/iovs.11-8002

ARVO Annual Meeting Abstract April 2011

Involvement Of Both VEGFR1 And R2 In Retinal Neovascularization (NV) And Vascular Permeability

Hu Huang; Stanley A. Vinores

Invest. Ophthalmol. Vis. Sci.. 2011; 52(14):4002.

ARVO Annual Meeting Abstract April 2011

Retinal And Choroidal Neovascularization, Blood-retinal Barrier Breakdown, And Apoptosis Are Inhibited In The Absence Of Prolyl Hydroxylase-1

Stanley A. Vinores; Hu Huang; Sara Van de Veire; Mansi Dalal; Richard Semba; Peter Carmeliet

Invest. Ophthalmol. Vis. Sci.. 2011; 52(14):1792.

Retina March 2011

TNFα Is Required for Late BRB Breakdown in Diabetic Retinopathy, and Its Inhibition Prevents Leukostasis and Protects Vessels and Neurons from Apoptosis

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Hu Huang; Jarel K. Gandhi; Xiufeng Zhong; Yanhong Wei; Junsong Gong; Elia J. Duh; Stanley A. Vinores
TAGS: apoptosis, diabetes mellitus, diabetes mellitus, type 2, diabetic retinopathy, leukostasis, mice, tumor necrosis factor-alpha, neurons
Invest. Ophthalmol. Vis. Sci.. 2011; 52(3):1336-1344. https://doi.org/10.1167/iovs.10-5768

Includes: Supplemental Content

Physiology and Pharmacology November 2010

Parstatin Suppresses Ocular Neovascularization and Inflammation

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Hu Huang; Panagiotis Vasilakis; Xiufeng Zhong; Ji-Kui Shen; Katerina Geronatsiou; Helen Papadaki; Michael E. Maragoudakis; Sotirios P. Gartaganis; Stanley A. Vinores; Nikos E. Tsopanoglou
TAGS: corneal neovascularization, inflammation, mice, neovascularization, pathologic, retinal neovascularization, choroidal neovascularization, burns, peptides, leukostasis, vascular endothelial growth factor a
Invest. Ophthalmol. Vis. Sci.. 2010; 51(11):5825-5832. https://doi.org/10.1167/iovs.10-5576

ARVO Annual Meeting Abstract April 2010

The Effects of Overexpression of rAav9-vegf-b167 on Ocular Angiogenesis, Inflammation, and Blood-Retinal Barrier

X. Zhong; H. Huang; J.-K. Shen; L. Zentilin; M. Giacca; S. Vinores

Invest. Ophthalmol. Vis. Sci.. 2010; 51(13):4492.

ARVO Annual Meeting Abstract April 2010

TNF is Essential for Progressive BRB Breakdown in Diabetic Retinopathy and Oxygen-Induced Ischemic Retinopathy and It Promotes Apoptosis in Retinal Vessels and Neurons

S. A. Vinores; H. Huang; J. Gandhi; X. Zhong; W. Xiao

Invest. Ophthalmol. Vis. Sci.. 2010; 51(13):3826.

ARVO Annual Meeting Abstract April 2010

The Anti-plgf Antibody (5d11d4) Specifically Reduces Choroidal Neovascularization in vivo on a Mouse Model

S. van de veire; T. Van Bergen; M. Mazzone; B. Jonckx; S. A. Vinores; L. Moons; I. Stalmans; P. Carmeliet

Invest. Ophthalmol. Vis. Sci.. 2010; 51(13):4510.

ARVO Annual Meeting Abstract April 2010

Effects of Parstatin on Ocular Angiogenesis, Inflammation, and BRB Breakdown

H. Huang; P. Vasilakis; J.-K. Shen; K. Geronatsiou; H. Papadaki; J. L. Strande; M. E. Maragoudakis; S. P. Gartaganis; S. A. Vinores; N. E. Tsopanoglou

Invest. Ophthalmol. Vis. Sci.. 2010; 51(13):2808.

ARVO Annual Meeting Abstract April 2009

Altered Basement Membrane Composition and Increased Vascular Permeability in Diabetic Retinopathy

A. Chronopoulos; K. Trudeau; S. Roy; H. Huang; S. Vinores; S. Roy

Invest. Ophthalmol. Vis. Sci.. 2009; 50(13):26.

ARVO Annual Meeting Abstract May 2008

Anti-PlGF (Placental Growth Factor) Reduces Choroidal Neovascularization in a Mouse Model of Age-Related Macular Degeneration (ARMD)

S. Van de Veire; S. Vinores; A. Peric; M. Mazzone; G. Moons; J.-M. Stassen; A. Noel; I. Stalmans; P. Carmeliet

Invest. Ophthalmol. Vis. Sci.. 2008; 49(13):5411.

ARVO Annual Meeting Abstract May 2008

Prolonged Blockade of VEGF Family Members Does Not Cause Identifiable Damage to Retinal Neurons or Vessels

S. Ueno; M. E. Pease; D. M. B. Wersinger; T. Masuda; S. A. Vinores; D. J. Zack; H. A. Quigley; E. Keshet; P. A. Campochiaro

Invest. Ophthalmol. Vis. Sci.. 2008; 49(13):1381.

ARVO Annual Meeting Abstract May 2006

TNF Is Critical for Retinal Leukostasis Mediated by VEGF, IL–1ß, and PAF and for Blood–Retinal Barrier (BRB) Breakdown in Ischemic Retinopathy

S.A. Vinores; W.–H. Xiao; J. Shen; P.A. Campochiaro

Invest. Ophthalmol. Vis. Sci.. 2006; 47(13):1723.

ARVO Annual Meeting Abstract May 2006

Retinal Vascular Permeability Is Regulated by VEGFR1 and VEGFR2

W. Xiao; S.A. Vinores; D. Collen; P. Carmeliet; P.A. Campochiaro

Invest. Ophthalmol. Vis. Sci.. 2006; 47(13):503.

ARVO Annual Meeting Abstract May 2004

HIF–1 induced VEGF augments, but is not essential for normal retinal vascular development

S.A. Vinores; W.–H. Xiao; S.F. Hackett; H. Liu; P. Carmeliet; P.A. Campochiaro

Invest. Ophthalmol. Vis. Sci.. 2004; 45(13):4652.

ARVO Annual Meeting Abstract May 2004

VEGF and PDGFs Have Unique Effects on Ocular Vascular Development in Transgenic Mice

W.–H. Xiao; R. Abakah; J.D. Ash; S.A. Vinores

Invest. Ophthalmol. Vis. Sci.. 2004; 45(13):1908.

ARVO Annual Meeting Abstract May 2003

Mouse Model of Post-Surgical Breakdown of the Blood-Retinal Barrier

H. Liu; W. Xiao; A. Demetriades; S. Vinores; P. Campochiaro

Invest. Ophthalmol. Vis. Sci.. 2003; 44(13):353.

ARVO Annual Meeting Abstract May 2003

Growth and Development of Myofibroblasts in Two Transgenic Mouse Models of Tractional Retinal Detachment

J.D. Ash; J.J. Tomasek; W. Xiao; S.A. Vinores

Invest. Ophthalmol. Vis. Sci.. 2003; 44(13):2964.

ARVO Annual Meeting Abstract May 2003

Ischemia Induces Blood-retinal Barrier (BRB) Breakdown through HIF-1 Dependent VEGF Upregulation and VEGF Mediates BRB Breakdown through ICAM-1 and Adenosine

W. Xiao; S.A. Vinores; J. Shen; P. Carmeliet; P.A. Campochiaro

Invest. Ophthalmol. Vis. Sci.. 2003; 44(13):3209.

ARVO Annual Meeting Abstract May 2003

Immunocytochemical and Ultrastructural Comparison of Postnatal Eyes from VEGF, PDGF-A, and PDGF-B Transgenic Mice

S.A. Vinores; W. Xiao; T. Hudish; R. Abakah; L.W. Reneker; J.D. Ash

Invest. Ophthalmol. Vis. Sci.. 2003; 44(13):4966.

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Reduced Retinal Neovascularization, Vascular Permeability, and Apoptosis in Ischemic Retinopathy in the Absence of Prolyl Hydroxylase-1 Due to the Prevention of Hyperoxia-Induced Vascular Obliteration

Involvement Of Both VEGFR1 And R2 In Retinal Neovascularization (NV) And Vascular Permeability

Retinal And Choroidal Neovascularization, Blood-retinal Barrier Breakdown, And Apoptosis Are Inhibited In The Absence Of Prolyl Hydroxylase-1

TNFα Is Required for Late BRB Breakdown in Diabetic Retinopathy, and Its Inhibition Prevents Leukostasis and Protects Vessels and Neurons from Apoptosis

Parstatin Suppresses Ocular Neovascularization and Inflammation

The Effects of Overexpression of rAav9-vegf-b167 on Ocular Angiogenesis, Inflammation, and Blood-Retinal Barrier

TNF is Essential for Progressive BRB Breakdown in Diabetic Retinopathy and Oxygen-Induced Ischemic Retinopathy and It Promotes Apoptosis in Retinal Vessels and Neurons

The Anti-plgf Antibody (5d11d4) Specifically Reduces Choroidal Neovascularization in vivo on a Mouse Model

Effects of Parstatin on Ocular Angiogenesis, Inflammation, and BRB Breakdown

Altered Basement Membrane Composition and Increased Vascular Permeability in Diabetic Retinopathy

Anti-PlGF (Placental Growth Factor) Reduces Choroidal Neovascularization in a Mouse Model of Age-Related Macular Degeneration (ARMD)

Prolonged Blockade of VEGF Family Members Does Not Cause Identifiable Damage to Retinal Neurons or Vessels

TNF Is Critical for Retinal Leukostasis Mediated by VEGF, IL–1ß, and PAF and for Blood–Retinal Barrier (BRB) Breakdown in Ischemic Retinopathy

Retinal Vascular Permeability Is Regulated by VEGFR1 and VEGFR2

HIF–1 induced VEGF augments, but is not essential for normal retinal vascular development

VEGF and PDGFs Have Unique Effects on Ocular Vascular Development in Transgenic Mice

Mouse Model of Post-Surgical Breakdown of the Blood-Retinal Barrier

Growth and Development of Myofibroblasts in Two Transgenic Mouse Models of Tractional Retinal Detachment

Ischemia Induces Blood-retinal Barrier (BRB) Breakdown through HIF-1 Dependent VEGF Upregulation and VEGF Mediates BRB Breakdown through ICAM-1 and Adenosine

Immunocytochemical and Ultrastructural Comparison of Postnatal Eyes from VEGF, PDGF-A, and PDGF-B Transgenic Mice

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