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Cornea June 2013

Angiopoietin-Like Protein 2 Is a Potent Hemangiogenic and Lymphangiogenic Factor in Corneal Inflammation

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Tetsuya Toyono; Tomohiko Usui; Seiichi Yokoo; Mikiko Kimakura; Suguru Nakagawa; Satoru Yamagami; Keishi Miyata; Yuichi Oike; Shiro Amano
TAGS: angiopoietins, cornea, keratitis, lymphangiogenesis, proteins, sutures, mice, immunohistochemistry, rna, small interfering
Invest. Ophthalmol. Vis. Sci.. 2013; 54(6):4278-4285. doi: https://doi.org/10.1167/iovs.12-11497

ARVO Annual Meeting Abstract March 2012

Association of Angiopoietin-like Protein 2 with Inflammatory Signals in the Human Retinal Pigment Epithelium

Manabu Hirasawa; Motoyo Endo; Seiji Miyake; Toshio Narimatsu; Shunsuke Kubota; Misa Suzuki; Susumu Ishida; Kazuo Tsubota; Yuichi Oike; Yoko Ozawa

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):1655. doi:

ARVO Annual Meeting Abstract March 2012

Angptl2 Knockout Mice Suppress Inflammation In Retinal Vessels Induced By A High-fat Diet

Yasuhiro Ito; Yuichi Oike; Hidenobu Tanihara

Invest. Ophthalmol. Vis. Sci.. 2012; 53(14):3331. doi:

Retinal Cell Biology July 2009

Prevention of Ocular Inflammation in Endotoxin-Induced Uveitis with Resveratrol by Inhibiting Oxidative Damage and Nuclear Factor–κB Activation

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Shunsuke Kubota; Toshihide Kurihara; Hiroshi Mochimaru; Shingo Satofuka; Kousuke Noda; Yoko Ozawa; Yuichi Oike; Susumu Ishida; Kazuo Tsubota
TAGS: resveratrol, eye inflammation, uveitis, endotoxins, prevention
Invest. Ophthalmol. Vis. Sci.. 2009; 50(7):3512-3519. doi: https://doi.org/10.1167/iovs.08-2666

Retinal Cell Biology April 2008

Inhibition of Choroidal Neovascularization with an Anti-Inflammatory Carotenoid Astaxanthin

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Kanako Izumi-Nagai; Norihiro Nagai; Kazuhiro Ohgami; Shingo Satofuka; Yoko Ozawa; Kazuo Tsubota; Shigeaki Ohno; Yuichi Oike; Susumu Ishida
TAGS: carotenoids, choroidal neovascularization, anti-inflammatory agents, macrophages
Invest. Ophthalmol. Vis. Sci.. 2008; 49(4):1679-1685. doi: https://doi.org/10.1167/iovs.07-1426

Retinal Cell Biology September 2007

Eicosapentaenoic Acid Is Anti-Inflammatory in Preventing Choroidal Neovascularization in Mice

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Takashi Koto; Norihiro Nagai; Hiroshi Mochimaru; Toshihide Kurihara; Kanako Izumi-Nagai; Shingo Satofuka; Hajime Shinoda; Kousuke Noda; Yoko Ozawa; Makoto Inoue; Kazuo Tsubota; Yuichi Oike; Susumu Ishida
TAGS: anti-inflammatory agents, choroidal neovascularization, mice, eicosapentaenoic acid, peroxisome proliferator-activated receptors, choroid, molecule
Invest. Ophthalmol. Vis. Sci.. 2007; 48(9):4328-4334. doi: https://doi.org/10.1167/iovs.06-1148

Retinal Cell Biology September 2007

Suppression of Diabetes-Induced Retinal Inflammation by Blocking the Angiotensin II Type 1 Receptor or Its Downstream Nuclear Factor-κB Pathway

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Norihiro Nagai; Kanako Izumi-Nagai; Yuichi Oike; Takashi Koto; Shingo Satofuka; Yoko Ozawa; Kenji Yamashiro; Makoto Inoue; Kazuo Tsubota; Kazuo Umezawa; Susumu Ishida
TAGS: diabetes mellitus, diabetes mellitus, type 2, inflammation, intercellular adhesion molecule 1, receptor, angiotensin, type 1, vascular endothelial growth factor a, leukocytes, retina
Invest. Ophthalmol. Vis. Sci.. 2007; 48(9):4342-4350. doi: https://doi.org/10.1167/iovs.06-1473

Retinal Cell Biology May 2007

Suppression of Choroidal Neovascularization by Inhibiting Angiotensin-Converting Enzyme: Minimal Role of Bradykinin

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Norihiro Nagai; Yuichi Oike; Kanako Izumi-Nagai; Takashi Koto; Shingo Satofuka; Hajime Shinoda; Kousuke Noda; Yoko Ozawa; Makoto Inoue; Kazuo Tsubota; Susumu Ishida
TAGS: choroidal neovascularization, angiotensin-converting enzyme, bradykinin, vascular endothelial growth factor a, intercellular adhesion molecule 1, monocyte chemoattractant protein-1
Invest. Ophthalmol. Vis. Sci.. 2007; 48(5):2321-2326. doi: https://doi.org/10.1167/iovs.06-1296

Retinal Cell Biology January 2007

Role of Nonproteolytically Activated Prorenin in Pathologic, but Not Physiologic, Retinal Neovascularization

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Shingo Satofuka; Atsuhiro Ichihara; Norihiro Nagai; Takashi Koto; Hajime Shinoda; Kousuke Noda; Yoko Ozawa; Makoto Inoue; Kazuo Tsubota; Hiroshi Itoh; Yuichi Oike; Susumu Ishida
TAGS: intercellular adhesion molecule 1, ischemia, retinal diseases, retinal neovascularization, vascular endothelial growth factor a, vascular endothelial growth factor receptor-2, vegfr-1, mice, proteins
Invest. Ophthalmol. Vis. Sci.. 2007; 48(1):422-429. doi: https://doi.org/10.1167/iovs.06-0534

Retinal Cell Biology December 2006

Neuroprotective Effects of Angiotensin II Type 1 Receptor (AT1R) Blocker, Telmisartan, via Modulating AT1R and AT2R Signaling in Retinal Inflammation

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Toshihide Kurihara; Yoko Ozawa; Kei Shinoda; Norihiro Nagai; Makoto Inoue; Yuichi Oike; Kazuo Tsubota; Susumu Ishida; Hideyuki Okano
TAGS: inflammation, signal transduction, telmisartan, receptor, angiotensin, type 1, synaptophysin
Invest. Ophthalmol. Vis. Sci.. 2006; 47(12):5545-5552. doi: https://doi.org/10.1167/iovs.06-0478

Retinal Cell Biology June 2006

Suppression of Ocular Inflammation in Endotoxin-Induced Uveitis by Inhibiting Nonproteolytic Activation of Prorenin

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Shingo Satofuka; Atsuhiro Ichihara; Norihiro Nagai; Kenji Yamashiro; Takashi Koto; Hajime Shinoda; Kousuke Noda; Yoko Ozawa; Makoto Inoue; Kazuo Tsubota; Fumiaki Suzuki; Yuichi Oike; Susumu Ishida
TAGS: endotoxins, eye inflammation, intercellular adhesion molecule 1, interleukin-6, leukocytes, monocyte chemoattractant protein-1, proteins, uveitis, leukocyte cell adhesion
Invest. Ophthalmol. Vis. Sci.. 2006; 47(6):2686-2692. doi: https://doi.org/10.1167/iovs.05-1458

Retina August 2005

Suppression of Ocular Inflammation in Endotoxin-Induced Uveitis by Blocking the Angiotensin II Type 1 Receptor

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Norihiro Nagai; Yuichi Oike; Kousuke Noda; Takashi Urano; Yoshiaki Kubota; Yoko Ozawa; Hajime Shinoda; Takashi Koto; Kei Shinoda; Makoto Inoue; Kazuo Tsubota; Kenji Yamashiro; Toshio Suda; Susumu Ishida
TAGS: endotoxins, eye inflammation, intercellular adhesion molecule 1, receptor, angiotensin, type 1, telmisartan, uveitis, proteins, mice, leukocytes
Invest. Ophthalmol. Vis. Sci.. 2005; 46(8):2925-2931. doi: https://doi.org/10.1167/iovs.04-1476

Retinal Cell Biology March 2005

Selective Suppression of Pathologic, but Not Physiologic, Retinal Neovascularization by Blocking the Angiotensin II Type 1 Receptor

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Norihiro Nagai; Kousuke Noda; Takashi Urano; Yoshiaki Kubota; Hajime Shinoda; Takashi Koto; Kei Shinoda; Makoto Inoue; Takayuki Shiomi; Eiji Ikeda; Kazuo Tsubota; Toshio Suda; Yuichi Oike; Susumu Ishida
TAGS: intercellular adhesion molecule 1, ischemia, receptor, angiotensin, type 1, retinal diseases, retinal neovascularization, telmisartan, vegfr-1, retina, mice
Invest. Ophthalmol. Vis. Sci.. 2005; 46(3):1078-1084. doi: https://doi.org/10.1167/iovs.04-1101

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Angiopoietin-Like Protein 2 Is a Potent Hemangiogenic and Lymphangiogenic Factor in Corneal Inflammation

Association of Angiopoietin-like Protein 2 with Inflammatory Signals in the Human Retinal Pigment Epithelium

Angptl2 Knockout Mice Suppress Inflammation In Retinal Vessels Induced By A High-fat Diet

Prevention of Ocular Inflammation in Endotoxin-Induced Uveitis with Resveratrol by Inhibiting Oxidative Damage and Nuclear Factor–κB Activation

Inhibition of Choroidal Neovascularization with an Anti-Inflammatory Carotenoid Astaxanthin

Eicosapentaenoic Acid Is Anti-Inflammatory in Preventing Choroidal Neovascularization in Mice

Suppression of Diabetes-Induced Retinal Inflammation by Blocking the Angiotensin II Type 1 Receptor or Its Downstream Nuclear Factor-κB Pathway

Suppression of Choroidal Neovascularization by Inhibiting Angiotensin-Converting Enzyme: Minimal Role of Bradykinin

Role of Nonproteolytically Activated Prorenin in Pathologic, but Not Physiologic, Retinal Neovascularization

Neuroprotective Effects of Angiotensin II Type 1 Receptor (AT1R) Blocker, Telmisartan, via Modulating AT1R and AT2R Signaling in Retinal Inflammation

Suppression of Ocular Inflammation in Endotoxin-Induced Uveitis by Inhibiting Nonproteolytic Activation of Prorenin

Suppression of Ocular Inflammation in Endotoxin-Induced Uveitis by Blocking the Angiotensin II Type 1 Receptor

Selective Suppression of Pathologic, but Not Physiologic, Retinal Neovascularization by Blocking the Angiotensin II Type 1 Receptor

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